Ive had this happen to me tons of times. I wake up, and cant move a muscle, I cant even breathe. At the same time, I see a shadowy figure out the corner of my eye. No matter what I do, I cant move. A few times, I mangaged to moan out for help - hoping someone will wake me up. But so far, no one has heard me.
My question is - what exactly is the old hag? Is it a result of diet? Or is it a way to experience one of those paranormal things on the forums (lucid dreams, out of body experiences, etc, etc)
If it IS the start of something paranormal. What can I do for it to continue along the paranormal path?
Well for one thing, Lucid Dreaming IS NOT paranormal. Don't confuse the two.
The old hag is a hallucination. Even if you were on a diet, the hag isn't the result of one. It looks like you saw her through Sleep Paralysis (No Shift, this doesn't look like REM Atonia. ).
EDIT: Don't know why, but I move this to Sleep and Health. It sure doesn't fit in Dream Control. Feel free to move this accordingly fellow DGs. I blame sickness for this move.
Last edited by Snowy Egypt; 01-21-2009 at 04:51 AM.
This is not paranormal or otherworldly, and there is no need for such drastic interpretations. Sleep Paralysis is simply the mechanism of REM atonia (your body paralyzing itself during REM sleep so that you don't act out your dreams) happening while you are awake. It's not anything paranormal, it's just the simple paralysis of your body. As a disorder, it happens more frequently than in the normal populations, some of whom experience it rarely throughout their lives.
Sleep paralysis is sometimes accompanied by hallucinations, just like hypnagogic and hypnopompic hallucinations, but which can be infinitely more terrifying and they think that this is simply because of the result of the overwhelming fear a person can have because they are paralyzed and hallucinating. Don't worry. You're not possessed. You simply have a simple problem that many people before you have had, and many people after you will have, and that the normal population may sometimes experience.
Snowy, I think this was the appropriate place to put this since he is speaking of a disorder, isolated sleep paralysis. And yea... why the hell would it be REM atonia?
Spoiler for more than you ever wanted to know about SP:
SP is a transient, conscious state of involuntary immobility occurring immediately prior to falling asleep or upon wakening and is classified as a parasomnia associated with REM (ASDA, 1990). Although individuals are unable to make gross bodily movements during SP they are able to open their eyes and subsequently to report accurately on events in their surroundings during the episode (Hishikawa & Kaneko, 1965). Approximately 25 to 40% of people report some SP experience (Cheyne, Newby-Clark, & Rueffer, in press; Fukuda, Ogilvie, Chilcott, Vendittelli, & Takeuchi, 1998; Spanos, McNulty, DuBreuil, Pires, & Burgess, 1995), although the incidence may vary across cultures (Fukuda, Miyasita, & Ishihara, 1987; Ness, 1978).
SP has traditionally been linked with narcolepsy and cataplexy as part of the ‘‘narcoleptic tetrad,’’ but is considerably more common than the latter disorders, the incidence of which range from .03 to .16% (Hishikawa & Shimizu, 1995). A number of anomalous sensory experiences frequently accompany SP. In the present study, these are referred to, collectively, as hypnagogic and hypnopompic experiences (HHEs). The HHEs include an acute sense of a monitoring ‘‘evil presence,’’ combinations of auditory and visual hallucinations, pressure on the chest, as well as suffocating, choking, floating, out-of-body, and flying sensations (Hishikawa, 1976; Hufford, 1982). Although these experiences bear some similarity to non-SPrelated pre- and postdormital hypnagogic images and sensations (Foulkes & Vogel, 1965; Mavromatis, 1987; Rowley, Stickgold, & Hobson, 1998; Schacter, 1976), HHEs accompanying SP appear to be substantially more vivid, elaborate, multimodal, and terrifying (Hufford, 1982; Takeuchi, Miyasita, Inugami, Sasaki, & Fukuda, 1994).
It has been conjectured that complex combinations of SP-related HHEs form the basis of diverse worldwide cultural accounts of nocturnal incubus/succubus assaults, spirit possessions, old hag attacks, ghostly visitations, and alien abductions (Adler, 1994; Bloom & Gelardin, 1976; Firestone, 1985; Fukuda, 1989; Hufford, 1976, 1982; Liddon, 1967; Ness, 1978; Wing, Lee, & Chen, 1994). In these accounts, a dreadful and evil presence in the form of a vampiric lamia, demon, spirit, or hag sits on the victim’s chest and smothers or chokes the helpless sleeper.
SP has been experimentally linked to REM states, particularly with sleep-onset and sleep-offset REM (Hishikawa & Kaneko, 1965; Nan’no, Hishikawa, & Koida,
1970). Hishikawa and Shimizu (1995) speculate that SP may be produced by hyperactivation of cholinoceptive and/or cholinergic Sleep-on neural populations or, they deem more likely, hypoactivation of noradrenergic or serotonergic Sleep-off populations in the pons. Thus, SP may reflect an anomaly of the functioning of the monoaminergic systems and/or their inhibition of cholinergic systems (Hishikawa & Shimizu, 1995). Sensory thresholds for awakening are relatively high during REM, suggesting that there may be, at best, weak and inconsistent cortical sensory processing during REM (Llina´s & Pare´, 1991). REM associated with the night-mare, however, appears to differ from dream-related REM in that there is little or no blocking of exteroceptive stimulation and no loss of waking consciousness (Hishikawa, 1976; Hishikawa & Kaneko, 1965). In any case, the throughput of sensory information along thalamocortical pathways during REM may be quite variable and, at times, exceed that during waking states (Inoue, Duysens, Vosser, & Coenen, 1993; van Hulzen & Coenen, 1984). During phasic SP, periods of high thalamic ‘‘transfer ratio’’ (Coenen & Vendrick, 1972) may result in high levels of both exteroceptive input and quasi-random activation originating in the brain stem. A major and distinctive feature of SP, we will argue, is the anomalous combination of high levels of exogenous and endogenous sources of cortical activation. Finally, the immobility of SP is also consistent with the general atonia maintained during REM by marked and sustained hyperpolarization of the spinal motoneurons (Chase & Morales, 1989). (1)
Sleep paralysis occurs immediately prior to falling asleep or upon waking. During these episodes, individuals are conscious of their surroundings and able to open their eyes, but unable to move (Hishikawa 1976), An acute sense of fear and various hypnagogic and hypnopompic experiences often accompany sleep paralysis, although little systematic evidence is available
on the prevalence of different experiences within a sleep paralysis episode.
Sleep paralysis may occur during sleep onsetor offset-REM (Fukuda 1994; Hishikawa and Shimizu 1995) and the hallucinoid experiences may result from neurological events associated with REM dream imagery (Hishikawa and
Shimizu 1995), These experiences include a sensed presence, auditory and visual hallucinations, unusual bodily sensations including floating, and feelings of pressure on the body (Hishikawa 1976; Hufford 1982),
The Waterloo Sleep Experiences Scale was designed to assess the prevalence of sleep paralysis and associated experiences, as well as to permit the analysis of relations among such experiences. Related research with this instrument has discovered a high degree of structure in the
patterning of hypnagogic and hypnopompic experiences (Cheyne et al. in press). This structure is consistent with cultural accounts of the 'Old Hag', 'Kanashibari', and ghost oppression that have been linked to sleep paralysis and associated experiences...
In our investigation, one-quarter of respondents who endorsed the basic paralysis question reported no additional hallucinatory features, and about 5% of the entire sample reported experiencing the full range of hallucinoid experiences associated with cultural phenomena such as the 'Old Hag.'
This latter finding suggests that instruments that fail to assess hallucinoid experiences along with prevalence rates of sleep paralysis may be misleading.
For example, reports of 30-40% in the literature may give the false impression that very large proportions of the population are experiencing the equivalent ofthe 'Old Hag' phenomenon (Hufford 1982; Ness 1978). The results also indicate that fear is associated with the reporting of hallucinoid experiences over and above the occurrence of sleep paralysis itself. Thus, at least some degree of fear is a response to something more than paralysis itself.
Indirect evidence was provided for a model in which sensed presence during sleep paralysis is associated with a fear reaction, possibly associated with amygdaloid and temporal lobe activation (Cheyne et al. in press) which, in turn, motivates additional experiences. Sensed presence and associated fear
may increase vigilance, detection, and interpretation of both endogenous (perhaps involving REM-related, oculomotor, middle ear, and primary sensory cortical activation) and exogenous environmental events. A sensed presence associated with fear may also initiate a search for an external threat and
focus attention on information in the visual and auditory systems. Visual experiences in particular may 'flesh out' the sense of presence and give it apparent form and substance.
The sense of a disembodied presence may subsequently fade and be replaced with more substantive perceptual experiences. This may explain why, in contrast to some of the other hallucinoid experiences, sensed presence was somewhat less frequent in the most elaborate reports than in the sparser accounts. (2)
1. Hypnagogic and Hypnopompic Hallucinations during Sleep Paralysis: Neurological and Cultural Construction of the Night-Mare
J. Allan Cheyne, Steve D. Rueffer, and Ian R. Newby-Clark 2. Relations among hypnagogic and hypnopompic experiences associated with sleep paralysis*
J, ALLAN CHEYNE, IAN R. NEWBY-CLARK andSTEVE D, RUEFFER
Your body likes to be retarded sometimes, and will on occasion actually resist your intended movement, as opposed to just not working. The demon is a result of not getting enough cake as a child, or something. Isn't psychology great?
Before I go back and read the replies in-depth, I should clarify my paranormal comment.
I've seen a lot of abbrviations around this forum. LD, WILD, DILD DIELD, etc. I know that a lucid dream is just an intense dream, but I didnt know if the other abbreviations were for something beyond dreams.
Edit: I finally had some time to sit down & dig around the site. Understand what most of the various abbreviations mean. So I guess the paranormal comment can be ignored. Just chalk it up to be being new around here.
Last edited by rocket70433; 01-22-2009 at 05:08 AM.
Thanks shift -
I knew that there really isnt the old hag there. I didn't even know it was called old hag until very recently. I was trying to personify sleep paralysis.
I remember one of my first experiences with sleep paralysis, I thought Chucky (from Childs Play) was behind my back. There have been a few times that I just had the paralysis, without ever imagining any figure
I was just curious what was really causing it, and if I could somehow use it to my advantage. Or if there is something I could do to prevent it. (Like reduce the amount of caffine in my diet)
I'm new to this forum, and am trying to make sense out of what everyone is saying. But thanks for your info & help!!
I just wasn't sure how many of the abbreivated terms referred to different types of dreams, and what referred to astral projection & out of body experienes.
Thanks snowy - like I said in my edit, I finally understand what's going on here. I'll look around the new member thread to ask about that stuff some more.
Similar thing happened to me for the first time the other night except I think I was still in the dream. There was a shadowy figure in the corner (before this, I was in someone's house with an female accomplice. I was about to make 'a move' when there was someone at the window and it was a 'ghost' or something) and I was petrified. Couldn't move my body, I remember trying to say the 'Our Father' but I could barely move my mouth. Lasted a good 10-15 seconds
Oh and I don't know if this has anything to do with it but I had some cheese before I went to bed
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