Right now Im just trying to prove that this concept works, I will need an assistant most likely my brother who will stand on the side of my bed and when he sees my eye lids flickering he will know that im dreaming. Then he should put the ultrasonic massages over my upper forehead an will see what happens.
Im not sure if this will work, but I have no doubt that the reason why where not lucid is because the dorsolateral prefrontal cortex is deactivated in NREM and fails to reactivate in REM.
Read this article/study that explains that the self-awareness experienced in waking and its loss in dreaming is the result of the deactivation of the dorsolateral prefrontal cortex.
http://www.bostonneuropsa.org/PDF%20...ET_AL.__02.pdf
Heres some of the important points in the article.
"The differences between the self-awareness experienced in waking and its diminution in dreaming can be explained by deactivation of the dorsolateral prefrontal cortex during REM sleep. Here,we propose that this deactivation results from a direct inhibition of the dorsolateral prefrontal cortical neurons by acetylcholine, the release of which is enhanced during REM sleep."
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"As a consequence of deactivation of the dorsolateral prefrontal cortex (DLPFC) during sleep, executive functions such as self-consciousness and analytical thought are severely impaired in NREM sleep and are weak in REM sleep."
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"In summary, the changes associated with the transition from waking to NREM sleep might be ascribed to a globally reduced level of activity. This is
followed by the selective reactivation of the most posterior and medial prefrontal areas and continued deactivation of anterior and lateral portions at the onset of REM sleep."
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"The transition from waking to NREM is characterized by frontal deactivation as reported in positron emission tomography (PET) studies using either H2[15O] [15,16] or 18F deoxyglucose (18FDG) [17] and quantitative EEG studies[18–20]. Deactivation increases with the deepening of NREM sleep [14] and is maintained in the transition from NREM to REM sleep [21]. However, with the onset of REM sleep, portions of the ventromedial, limbic-related prefrontal cortex and closely associated medial subcortex and cortex are reactivated, sometimes to levels that exceed those of waking [22].
For example, in a H2[15O] PET study that specifically compares REM with NREM sleep, the prefrontal areas reactivated during REM include anterior cingulate (BA 32), caudal orbital and medial prefrontal (BA 10) cortices [13], but the DLPFC remained deactivated. Nofzinger et al. [23] have termed this frontal area the ‘anterior paralimbic REM activation area’ with the core structures being subgenual and pregenual anterior cingulate cortex, the amygdala and the insula."
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"At the beginning of the REM phase, however, activation of the acetylcholine system, starting from the pontine reticular formation, provokes a general increase in cortical activity [26]. We propose that the DLPFC remains relatively deactivated in REM sleep because it is directly inhibited by acetylcholine. Findings supporting this include a functional magnetic resonance study showing that acetylcholine-mediated enhancement
increased the activity of extrastriate cortex but decreased the activity of the anterior prefrontal cortex [27].
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"Taken together, these results indicate that the forebrain activation and synthesis processes that underlie dreaming, with selective activation
of the subcortical and cortical limbic structures (which mediate emotion) and relative inactivation of the DLPFC (which mediates directed thought), are very different from those during waking"
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"In this review, we have advanced the idea that the deactivation of the prefrontal cortex during REM sleep is the result of direct acetylcholine-mediated inhibition of this cortical region. According to our proposal, cholinergic pathways are responsible for the reactivation in REM sleep of the prefrontal limbic cortex, but not of the DLPFC, thus depriving dream mentation of logical reasoning capacities."
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